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𝐇𝐞𝐚𝐥𝐭𝐡𝐜𝐚𝐫𝐞 𝐀𝐜𝐪𝐮𝐢𝐫𝐞𝐝 𝐈𝐧𝐟𝐞𝐜𝐭𝐢𝐨𝐧𝐬 These are infections that patients acquire during treatment in a healthcare setting, such as a hospital, long-term care facility, or dialysis center. Hospital Acquired Infections (HAI) is the term used by CMS to describe HAIs acquired during certain medical and surgical procedures. These include surgeries of the heart and blood vessels, urinary tract/bladder, gastrointestinal tract, and respiratory system; open-heart surgery; dialysis; and selected skin and wound care procedures. ....#cancer #cancercare #chemocare #cancersymptoms #cancertreatment #chemotherapy #cancercure #cancercells Read more about this topic, in our blog: cancercelltreatment.com/2022/02/17/healthcare-associated-infection/ ... See MoreSee Less

1 hour ago  ·  

𝐓𝐡𝐞 𝐅𝐮𝐭𝐮𝐫𝐞 𝐨𝐟 𝐓𝐫𝐞𝐚𝐭𝐦𝐞𝐧𝐭𝐬 Targeted therapy is a treatment that uses drugs or other substances to identify and attack specific cancer cells without harming normal cells. Because targeted therapies are designed to hit specific targets on cancer cells, they are often called “targeted therapies”......#cancer #cancercare #chemocare #cancersymptoms #cancertreatment #chemotherapy #cancercure #cancercells cancercelltreatment.com/2022/03/07/cancer-new-paradigm/ ... See MoreSee Less

1 day ago  ·  

𝐌𝐢𝐧𝐞𝐫𝐚𝐥 𝐑𝐞𝐥𝐚𝐭𝐢𝐨𝐧𝐬𝐡𝐢𝐩𝐬 - 𝐕𝐢𝐭𝐚𝐦𝐢𝐧𝐬 - 𝐄𝐧𝐝𝐨𝐜𝐫𝐢𝐧𝐞𝐬 𝐚𝐧𝐝 𝐇𝐞𝐚𝐥𝐭𝐡 Nutrient interrelationships are complex, especially among trace the elements. A mineral cannot be affected without affecting at least two other minerals, each of which will then affect two others, etc. Mineral relationships can be compared to a series of intermeshing gears that are all connected, some directly and some indirectly. .....#cancer #cancercare #chemocare #cancersymptoms #cancertreatment #chemotherapy #cancercure #cancercells ... See MoreSee Less

2 days ago  ·  

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Nutrient interrelationships are complex, especially among the trace elements. A mineral cannot be effected without affecting at least two other minerals, each of which will then affect two others, etc.
#cancer #cancercare

https://elements.envato.com/colorful-minerals-rotates-and-pills-fall-down-CNDN737

𝐇𝐨𝐰 𝐦𝐢𝐧𝐝 𝐚𝐟𝐟𝐞𝐜𝐭𝐬 𝐛𝐨𝐝𝐲: 𝘔𝘦𝘯𝘵𝘢𝘭 𝘩𝘦𝘢𝘭𝘵𝘩 𝘪𝘴 𝘢𝘴 𝘪𝘮𝘱𝘰𝘳𝘵𝘢𝘯𝘵 𝘢𝘴 𝘱𝘩𝘺𝘴𝘪𝘤𝘢𝘭 𝘩𝘦𝘢𝘭𝘵𝘩. #mindfulness
#mindandbody #strongmind
https://cancercelltreatment.com/2022/03/28/sympathetic-stimulation/

𝐂𝐲𝐭𝐨𝐭𝐨𝐱𝐢𝐜 𝘤𝘢𝘯𝘤𝘦𝘳 𝘵𝘳𝘦𝘢𝘵𝘮𝘦𝘯𝘵𝘴 𝘢𝘳𝘦 𝘵𝘩𝘰𝘴𝘦 𝘵𝘩𝘢𝘵 𝘢𝘧𝘧𝘦𝘤𝘵 𝘵𝘩𝘦 𝘤𝘢𝘯𝘤𝘦𝘳 𝘤𝘦𝘭𝘭𝘴 𝘴𝘱𝘦𝘤𝘪𝘧𝘪𝘤𝘢𝘭𝘭𝘺 𝘸𝘪𝘵𝘩𝘰𝘶𝘵 𝘢𝘧𝘧𝘦𝘤𝘵𝘪𝘯𝘨 𝘰𝘵𝘩𝘦𝘳 𝘤𝘦𝘭𝘭𝘴 𝘪𝘯 𝘵𝘩𝘦 𝘣𝘰𝘥𝘺.
#cancer #cancertreatment
https://cancercelltreatment.com/2022/02/27/types-of-cancer-treatment/

Cancer cells have abnormal mutations in their DNA. These mutations allow cancer cells to grow and divide uncontrollably.
#cancer #cancercare #chemocare #cancersymptoms #cancertreatment #chemotherapy #cancercure #cancercells
https://cancercelltreatment.com/2022/04/18/cancer-cell-mutations/

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Tumor Microenvironment - The Environment at the Cellular Level

May 31, 2022
Est. Reading: 3 minutes

The tumor microenvironment is a complex and dynamic milieu that plays a pivotal role in tumor progression and metastasis. Tumor cells interact with various stromal cells, including cancer-associated fibroblasts (CAFs), myeloid-derived suppressor cells (MDSCs), and immune cells, to create a microenvironment that promotes tumor growth and metastasis.

This metastasis may lead to creating a malignant tumor or a benignant tumor. Benign typically means that it is a non-cancerous tumor. Tumor-educated leukocytes also contribute to the immunosuppressive microenvironment. A better understanding of the interactions between tumor cells and their microenvironment will provide insights into developing novel therapeutic strategies for cancer treatment.

Tumor Environment at the cellular level

Tumor cells produce numerous soluble factors, such as cytokines, chemokines, and growth factors, that promote tumor growth and metastasis by altering the microenvironment. Tumor-derived cytokines, such as TGF-β, IL-6, and VEGF, stimulate the proliferation and differentiation of CAFs. TGF-β also inhibits the function of immune cells, including T cells and natural killer (NK) cells. Tumor-derived chemokines, such as CCL2, CXCL8, and CXCL12, attract leukocytes to the tumor site and promote tumor progression. Tumor-derived growth factors, such as IGF-1 and FGF-2, promote cell proliferation and angiogenesis.

Inflammation and Oxidative Stress at Cellular Level

In addition to the direct effects of tumor cells on the microenvironment, inflammation and oxidative stress also contribute to tumor progression. Inflammation is how the body responds to cellular injury or infection. This response releases immune cells' inflammatory mediators, such as cytokines, chemokines, and growth factors. These mediators promote cell proliferation, cell survival, angiogenesis, and metastasis.

Oxidative stress is a condition in which there is an imbalance between the production of reactive oxygen species (ROS) and the body's ability to detoxify them. ROS develops due to normal cellular metabolism or exposure to environmental stressors, such as UV radiation. This imbalance can lead to cell damage and death. Inflammation and oxidative stress contribute to tumor progression by altering the microenvironment to promote growth and metastasis.

Damaged and Mutated Cells at Cellular Level

Tumor cells are damaged and mutated cells at the cellular level. These changes happen due to various factors, including genetic mutations, exposure to environmental stressors, and inflammation. Damaged and mutated cells divide uncontrollably and do not die when they should—this division results in the formation of a mass of abnormal cells, known as a tumor. Tumors can be benign or malignant.

  • Benignant Tumor: A non-cancerous tumor that does not spread to other body parts.
  • Malignant Tumor: A cancerous tumor that can spread to other body parts.

Tumors can grow and spread in several ways. One way is to produce cytokines, chemokines, and growth factors that alter the tumor microenvironment to promote abnormal growth and metastasis. Another way is through inflammation and oxidative stress, which also contribute to tumor progression. Damaged and mutated cells at the cellular level are a significant cause of tumor growth and metastasis.

Risk Factors for Damaged and Mutated Cells at Cellular Level

There are numerous risk factors for developing damaged and mutated cells at Cellular Level. These include:

  • Genetic mutations: Damaged and genetic mutations can cause mutated cells. This damage can happen if there is a mutation in a gene that regulates cell growth or cell death. This mutation can lead to the formation of a tumor.
  • Exposure to environmental stressors: Damaged and mutated cells can also happen due to environmental stressors, such as UV radiation. This exposure can damage the DNA of cells and lead to the formation of a tumor.
  • Inflammation: Inflammation can also cause damaged and mutated cells. This damage happens because inflammation can damage DNA and promote the growth of cancerous cells.

Prevention of Damaged and Mutated Cells at the Cellular Level

There are several ways to prevent damaged and mutated cells at the cellular level. One way is to avoid exposure to environmental stressors, such as UV radiation. Another way is to prevent inflammation by eating a healthy diet and managing stress. Finally, a person can reduce their risk by getting regular checkups and screenings, which can help detect cancer early.

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